10. Two different mutant of Drosophilla gives a mutant black body color. When these mutants are crossed all progeny have wild type color, It means mutation are (1) Codominant (2) Allelic (3) Non Allelic (4) Epistatic

10. Two different mutant of Drosophilla gives a mutant black body color.  When these mutants are crossed all progeny have wild type color, It means mutation are
(1) Codominant        (2) Allelic
(3) Non Allelic          (4) Epistatic

Concept

  • Each parent fly is homozygous recessive for its own black‑body mutation.

  • Crossing mutant 1 (gene a) with mutant 2 (gene b) gives progeny that are heterozygous at both loci (a⁺a bb⁺ or similar).

  • Because each F₁ receives one functional (wild‑type) copy of each gene, pigment synthesis is restored and all offspring are wild type.

  • This is the classic result of a complementation test: mutations in different genes complement and give wild type. Those are called non‑allelic mutants.


Option‑wise explanation

  1. Codominant

    • Codominance means both alleles in a heterozygote are fully expressed (e.g., human AB blood group).

    • Here, each mutant alone is recessive; the F₁ appears wild type, not a mixture of black and wild. This is not codominance.

  2. Allelic

    • Allelic mutants affect the same gene.

    • If both parents are homozygous for different recessive alleles of the same gene (a¹a¹ × a²a²), the F₁ (a¹a²) still lacks any wild‑type copy and would remain mutant (black).

    • Because the cross gives wild type, the mutations are not allelic.

  3. Non allelic – correct

    • Non‑allelic = in different genes.

    • Each mutant has a defect in a different step of the same pathway; when combined, each supplies the wild‑type product missing in the other, so they complement and restore the wild‑type color.

  4. Epistatic

    • Epistasis is interaction where one gene’s allele masks or modifies the effect of another gene in specific genotype combinations.

    • While black body can involve epistatic interactions, the key observation here is full complementation (all F₁ wild). The defining conclusion from such a complementation test is that the mutations are in different genes, i.e., non‑allelic, not simply epistatic.

Therefore, the two mutant black‑body strains are non‑allelic mutants that complement each other.

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